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Effects of Delayed Treatment With Poly (ADP-Ribose) Polymerase Inhibitor After Ischemia-Reperfusion on Healing Phase of Renal Injury (Exhibit Hall, Orange County Convention Center, West Building) - Oct 13, 2022 - Abstract #KIDNEYWEEK2022KIDNEY_WEEK_1004; JPI-289 treatment of 0.5 and 0.75 mg/mL at 3 or 6 hours after hypoxia facilitated the proliferation of hypoxic HK-2 cells, whereas further treatment after 24 hours suppressed proliferation even with lower dosages. Conclusion Our results suggest that late treatment of PARP inhibitors after renal IRI did not have a beneficial effect on the recovery of ischemic AKI, but rather could have a negative effect on healing.
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[VIRTUAL] Poly (ADP-Ribose) Polymerase Inhibitor Treatment Attenuated Renal Ischemia-Reperfusion Injury (Virtual) - May 29, 2020 - Abstract #ATC2020ATC_1547; PARP inhibition with JPI-289 treatment showed favorable effects in ischemic AKI by attenuating intrarenal inflammatory cascade in a murine model and facilitating proliferation of hypoxic HK-2 cells. JPI-289 treatment mitigated renal injury by changing intrarenal immunologic micromilieu favorably in a murine ischemic AKI model and restored proliferation of HK-2 cells after hypoxia.
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Trial completion date, Trial primary completion date: Clinical Trial to Evaluate the Efficacy and Safety of JPI-289 in Patients With Acute Ischemic Stroke (clinicaltrials.gov) - Nov 18, 2019 P2a, N=110, Recruiting, JPI-289 treatment mitigated renal injury by changing intrarenal immunologic micromilieu favorably in a murine ischemic AKI model and restored proliferation of HK-2 cells after hypoxia. Trial completion date: Dec 2019 --> Dec 2021 | Trial primary completion date: Dec 2018 --> Dec 2020
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The Effects of PARP Inhibitor Treatment on Early Renal Injury in a Murine Ischemic AKI Model (Exhibit Hall, Walter E. Washington Convention Center) - Oct 14, 2019 - Abstract #KIDNEYWEEK2019KIDNEY_WEEK_3621; Conclusion JPI-289 treatment attenuated early renal injury in a murine ischemic AKI model and facilitated proliferation of hypoxic HK-2 cells. Further studies are needed to identify optimal dosage and administration timing of JPI-289 treatment.
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