- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: ADORA1 Promotes Hepatocellular Carcinoma Progression via PI3K/AKT Pathway. (Pubmed Central) - Dec 10, 2020 Treatment with PI3K inhibitor LY294002 blocked the effects of ADORA1 on tumor growth in either ADORA1-overexpressing or -deficiency cells...Treatment of ADORA1 antagonist oppositely suppressed HCC xenograft tumor growth. ADORA1 serves as an important oncoprotein and a promoter of cell proliferation through PI3K/AKT signaling pathway in hepatocellular carcinoma.
- |||||||||| quercetin (LY294002) / Eli Lilly, Tuoyi (toripalimab) / Shanghai Junshi Biosci, rapamycin topical (PTX-022) / Palvella Therapeutics, Ligand
Journal, PD(L)-1 Biomarker, IO Biomarker: Apelin-13 inhibits apoptosis and excessive autophagy in cerebral ischemia/reperfusion injury. (Pubmed Central) - Dec 5, 2020 Both LY294002 (20 μM) and rapamycin (500 nM), which are inhibitors of the PI3K/Akt/mTOR pathway, significantly attenuated the inhibition of autophagy and apoptosis caused by apelin-13...The study was approved by the Animal Ethical and Welfare Committee of Jining Medical University, China (approval No. 2018-JS-001) in February 2018.
- |||||||||| quercetin (LY294002) / Eli Lilly, Mozobil (plerixafor) / Sanofi, PD98059 / Wayne State University
Journal: Role of the stromal cell derived factor-1 in the biological functions of endothelial progenitor cells and its underlying mechanisms. (Pubmed Central) - Dec 5, 2020 Moreover, SDF-1 significantly increased the protein expression levels of phosphorylated (p)-Akt and p-ERK; however, SDF-1-induced effects on protein expression were suppressed by AMD3100, LY294002 and PD98059. Thus, SDF-1-induced EPC proliferation was mediated by activation of the Akt and ERK signaling pathways, whereas SDF-1-mediated EPC migration and tube formation only involved activation of the Akt signaling pathway.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Klotho gene improves oxidative stress injury after myocardial infarction. (Pubmed Central) - Dec 5, 2020 Further, the Klotho protein expression of the pDC316-Klotho group was significantly upregulated and the Nrf-2 and ARE proteins expressions of the LY294002 and pDC316-Klotho groups were significantly suppressed. Klotho overexpression improved findings of oxidative stress injury after myocardial infarction.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Cardioprotective effect exerted by Timosaponin BⅡ through the regulation of endoplasmic stress-induced apoptosis. (Pubmed Central) - Dec 1, 2020 Taken together, these results demonstrated that curcumin protects SH‑SY5Y cells against OGD/R injury by inhibiting apoptosis and oxidative stress, and via enhancing the APE1 level and activity, promoting PI3K/AKT pathway activation. These results suggested that TBⅡ protects against myocardial injury in vitro and enhances cellular defense capacity by inhibiting ER stress-mediated apoptosis pathways in vivo by activating the PI3K/Akt pathways.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Neural maturation enhanced by exercise-induced extracellular derivatives. (Pubmed Central) - Nov 26, 2020 Finally, the administration of exercise-induced EDs for 3 days increased the Histone 3 phosphorylation and β-III tubulin expression in Ink/Arf null neural stem cells and progenitors (NSPCs) under each proliferation and differentiation condition. These results suggest that exercise-induced circulating EDs may mediate neuronal maturation during exercise.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Endothelial Nitric Oxide Mediates the Anti-Atherosclerotic Action of Torenia concolor Lindley var. Formosama Yamazaki. (Pubmed Central) - Nov 25, 2020 Moreover, TCEE-induced NO production, and EC proliferation, migration, and tube formation were diminished by pretreatment with LY294002 (an Akt inhibitor), KN62 (a CaMKII inhibitor), and compound C (an AMPK inhibitor)...Moreover, chronic treatment with TCEE attenuated hyperlipidemia, systemic and aortic inflammatory response, and the atherosclerotic lesions in apolipoprotein E-deficient mice. Collectively, our findings suggest that TCEE may confer protection from atherosclerosis by preventing endothelial dysfunction.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal, IO Biomarker: CCN5 inhibits proliferation and promotes apoptosis of oral squamous cell carcinoma cells. (Pubmed Central) - Nov 21, 2020 Further, LY294002, a PI3K/AKT antagonist, was employed to explore the mechanism underlying the effects of CCN5 in the regulation of OSCC...Finally, xenograft analysis showed that CCN5 represses tumorigenesis of OSCC cells. These findings together suggest that CCN5 functions as a tumor suppressor for OSCC cell development through inactivation of PI3K/AKT signaling pathway, providing a potential candidate for OSCC therapy.
- |||||||||| sirolimus / Generic mfg.
Journal: Role of Lysophosphatidic Acid in Vascular Smooth Muscle Cell Proliferation. (Pubmed Central) - Nov 19, 2020 Furthermore, gene expressions for PLC β1 isoform, PKC δ and ε isoforms, casein kinase II β isoform and endothelin-1A receptors were elevated by LPA. These results suggest that the LPA-induced proliferation of VSM cells is mediated through the activation of GPCR and multiple protein kinases as well as gene expressions of their some specific isoforms.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal, IO Biomarker: Nano-silicon Dioxide Affects Apoptosis of Alveolar Macrophages via PI3K/AKt Signaling Pathway (Pubmed Central) - Nov 18, 2020 With LY294002 pretreatment, the mitochondrial transmembrane potential level and the expressions of p-PI3K, p-AKt and Bcl-2 were decreased, the expression of Bax and the apoptosis rate were increased. Our data suggested that the activation of PI3K/AKt signaling pathway played an important role in NS-induced apoptosis in alveolar macrophages.
- |||||||||| quercetin (LY294002) / Eli Lilly, U0126 / Promega, SP600125 / BMS
Journal, PARP Biomarker, IO Biomarker: Morusin induces apoptosis and autophagy via JNK, ERK and PI3K/Akt signaling in human lung carcinoma cells. (Pubmed Central) - Nov 12, 2020 A PI3K/Akt inhibitor (LY294002), a JNK inhibitor (SP60015) and a MEK/ERK inhibitor (U0126) contributed to the determination that these pathways were involved in both apoptosis and autophagy induced by morusin. Moreover, morusin treatment strikingly enhanced intracellular ROS level, an ROS scavenger NAC blocked cell death and changes of Akt, JNK and ERK induced by morusin.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal, PARP Biomarker: Capillarisin protects SH-SY5Y cells against bupivacaine-induced apoptosis via ROS-mediated PI3K/PKB pathway. (Pubmed Central) - Nov 6, 2020 Together, our results provide foundation for further development of safer and more effective glucocorticoid-based combination therapy of hematological malignancies using PI3K/Akt/mTOR inhibitors. Capillarisin protected SH-SY5Y cells against bupivacaine-induced apoptosis by inhibiting oxidative stress, mitochondrial injury, and endoplasmic reticulum stress via ROS-mediated of PI3K/PKB pathway.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Transcriptomics predicts compound synergy in drug and natural product treated glioblastoma cells. (Pubmed Central) - Nov 6, 2020 Furthermore, in combination, fucoxanthin synergizes with LY-294002 in inhibiting the growth of U87MG cells, suggesting complementarity in their molecular modes of action and pointing to further treatment combinations. The synergy we observe between the dietary nutraceutical fucoxanthin and the synthetic chemical LY-294002 in producing growth arrest in glioblastoma, illustrates the potential of nutri-pharmaceutical combinations in targeting this challenging disease.
- |||||||||| Ibrance (palbociclib) / Pfizer, quercetin (LY294002) / Eli Lilly, Verzenio (abemaciclib) / Eli Lilly
[VIRTUAL] Autophagy Inhibition Enhances CDK4/6 Inhibitor-Induced Apoptosis in t(8;21) Acute Myeloid Leukemia Cells (Poster Hall (Virtual Meeting)) - Nov 5, 2020 - Abstract #ASH2020ASH_807; Because CCND2 is not a druggable target, we examined the effect of CDK4/6 inhibitors (palbociclib and abemaciclib) on t(8;21) AML cells...Treatment of t(8;21) AML cells with the autophagy inhibitors chloroquine (CQ) or LY294002 in combination with abemaciclib significantly increased the frequency of apoptotic (Annexin V positive) cells compared with that in untreated cells, whereas CQ or LY294002 single treatment had no significant effect on apoptosis...These results suggest that autophagy inhibition enhances CDK4/6 inhibitor-induced apoptosis in t(8;21) AML cells. In conclusion, the present results indicate that inhibition of CDK4/6 and autophagy may be a novel and promising biomarker-driven therapeutic strategy for the treatment of t(8;21) AML.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Mitochondrial dysfunction increases pro-inflammatory cytokine production and impairs repair and corticosteroid responsiveness in lung epithelium. (Pubmed Central) - Nov 2, 2020 The reduced corticosteroid responsiveness may be mediated by glycolytic reprogramming, specifically glycolysis-associated PI3K signaling, as PI3K inhibitor LY294002 restored the sensitivity of CXCL8 secretion to corticosteroids in A549 Rho-0 cells. In conclusion, mitochondrial defects may lead to increased lung epithelial pro-inflammatory responses, reduced epithelial repair and reduced corticosteroid responsiveness in lung epithelium, thus potentially contributing to the pathogenesis of COPD.
- |||||||||| quercetin (LY294002) / Eli Lilly
Journal: Mechanical stress promotes biological functions of C2C12 myoblasts by activating PI3K/AKT/mTOR signaling pathway. (Pubmed Central) - Oct 28, 2020 The PI3K specific inhibitor LY294002 significantly decreased 4EBP1 expression and reduced p‑AKT and p‑mTOR expression compared with the mechanical stress group...In conclusion, mechanical stress promoted biological functions of C2C12 cells by activating the PI3K/AKT signaling pathway. These results may contribute to a better understanding of the effects of mechanical stress on cells.
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