- |||||||||| Journal: Redox Modulatory protective effects of ω-3 fatty acids rich fish oil against experimental colitis. (Pubmed Central) - May 9, 2019
The modulation of COX mediated pathways in UC-associated inflammation protein expressions of various pro-inflammatory cytokines such as TNF-α and enzymes of PG synthesis such as COX-2, PGES, TXAS and anti-inflammatory PGDS were observed. Refuting the earlier reports that suggested the contradictory effects of FO, in current study we evidently demonstrated that the protective effects of FO are mediated through molecular mechanisms involving the redox-regulation of metabolism of key lipid metabolites.
- |||||||||| Journal: Introduction to COX inhibitors. (Pubmed Central) - May 1, 2019
This effects in higher PGE₂ production, which in turn increases its capability to induce IL-1RI. No abstract available
- |||||||||| aspirin / Generic mfg.
Clinical, Journal, Gene Expression Profile: Regular aspirin use and gene expression profiles in prostate cancer patients. (Pubmed Central) - Apr 30, 2019 Other putative target genes had similar expression in tumors from aspirin users and non-users. If results are corroborated by experimental studies, a potential benefit of aspirin may be limited to a subset of prostate cancer patients.
- |||||||||| Review, Journal: Duchenne muscular dystrophy: Focus on arachidonic acid metabolites. (Pubmed Central) - Apr 19, 2019
5-LOX inhibition might be important for the survival of myofibers. Moreover H-PGDS inhibitors, cyclooxygenase (COX)-inhibiting NO donors (CINODs), inhibitors of Ca2+-independent PLA are some of the different pathways which can bring to further development of new drugs.
- |||||||||| celecoxib oral / generics
Journal, IO Biomarker: Cyclooxygenase-1 and -2 Play Contrasting Roles in Listeria-Stimulated Immunity. (Pubmed Central) - Apr 12, 2019 ...This phenotype was due to inhibition of the inducible COX-2 enzyme, as treatment with the COX-2-selective inhibitor celecoxib similarly inhibited the development of immunity...Taken together, our results suggest that COX-2 is necessary for optimal CD8 T cell responses to L. monocytogenes, whereas COX-1 is detrimental. Use of pharmacotherapies that spare COX-2 activity and the production of PGE like acetaminophen will be critical for the generation of optimal antitumor responses using L. monocytogenes.
- |||||||||| Journal: COX Inhibition Profiles and Molecular Docking Studies of the Lignan Hinokinin and Some Synthetic Derivatives. (Pubmed Central) - Apr 12, 2019
...The in vitro assays demonstrated dinitrohinokinin is about 13 times more selective for COX-2 than for COX-1, a similar profile observed for the drugs celecoxib (selective index ≈9) and meloxicam (selective index ≈11)...This lignan also interacts with COX-1 in a similar mode to meloxicam, blocking the access of the substrate to the catalytic cleft. Therefore, dinitrohinokinin is a promising lead for the design of selective COX-2 inhibitors.
- |||||||||| Zyflo CR (zileuton) / Vectura
Preclinical, Journal: Inhibition of 5-lipoxygenase by zileuton in a rat model of myocardial infarction. (Pubmed Central) - Apr 11, 2019 Studies were limited to a single NSAID (indomethacin), so future studies are needed to assess the generalizability of our findings and to establish a direct link to the human condition. 5-LOX inhibition in rat myocardial infarction model attenuated increased left ventricle NF-κB expression and apoptosis and these actions were not modulated by COX inhibitors.
- |||||||||| celecoxib oral / generics
Preclinical, Journal: Synthesis, in-vivo and in-vitro Anti-inflammatory Evaluation of some Novel Coumarin Derivatives. (Pubmed Central) - Apr 11, 2019 5-LOX inhibition in rat myocardial infarction model attenuated increased left ventricle NF-κB expression and apoptosis and these actions were not modulated by COX inhibitors. The newly synthesized ester derivatives 1-9 showed an improved activity compared with the parent compound III, in both in-vivo and in-vitro screening which proved that the new ester derivatives improve the activity and/or bioavailability.
- |||||||||| Journal: The antipyretic effect of paracetamol occurs independent of transient receptor potential ankyrin 1-mediated hypothermia and is associated with prostaglandin inhibition in the brain. (Pubmed Central) - Apr 3, 2019
The hypothermia induced by paracetamol was abolished in mice treated with the electrophile-scavenger N-acetyl cysteine. We conclude that paracetamol's antipyretic effect in mice is dependent on inhibition of cyclooxygenase activity, including the formation of pyrogenic prostaglandin E, whereas paracetamol-induced hypothermia likely is mediated by the activation of TRPA1 by electrophilic metabolites of paracetamol, similar to its analgesic effect in some experimental paradigms.-Mirrasekhian, E., Nilsson, J. L. Å., Shionoya, K., Blomgren, A., Zygmunt, P. M., Engblom, D., Högestätt, E. D., Blomqvist, A. The antipyretic effect of paracetamol occurs independent of transient receptor potential ankyrin 1-mediated hypothermia and is associated with prostaglandin inhibition in the brain.
- |||||||||| glibenclamide / generics
Preclinical, Journal: Endothelium-dependent and endothelium-independent vasorelaxant effects of tiliacorinine 12'-O-acetate and mechanisms on isolated rat aorta. (Pubmed Central) - Apr 3, 2019 However, this effect was not impacted by indomethacin (10 μM, cyclooxygenase inhibitor), tetraethylammonium (5 mM, K channel blocker), barium chloride (1 mM, K channel blocker), or glibenclamide (10 μM, K channel blocker)...The results of this study suggest that tiliacorinine 12'-O-acetate induced endothelium-dependent vasorelaxation through the eNOS/NO/sGC pathway, and also induced endothelium independent vasorelaxation involving the modulation of sGC activity, K channels, Ca influx through Ca channels and intracellular Ca release. The data concerning the benefits of tiliacorinine 12'-O-acetate might be further investigated for the application of tiliacorinine 12'-O-acetate as an antihypertensive compound.
- |||||||||| celecoxib oral / generics
Journal: Metabololipidomic profiling of functional immunoresolvent clusters and eicosanoids in mammalian tissues. (Pubmed Central) - Mar 27, 2019 ...The classic cyclooxygenase inhibitors, celecoxib and indomethacin, that block thromboxanes and prostanoids do not block production of the clot-driven SPM cluster...Thus, we identified a pro-resolving lipid mediator circuit and specific SPM cluster that promotes host defense. This new lipid mediator (LM)-SPM metabololipidomic approach now provides accessible metabolomic profiles in healthy and diseased human tissues, including cancer, for precision and personalized medicine.
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